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Warning: Clinical review pending: not for clinical use

Before you score. HAS-BLED does not preclude anticoagulation. A high score identifies correctable risk factors, not a contraindication.

For clinical decision support only. Always integrate with full clinical assessment, local guidelines, and patient preferences.

HAS-BLED Score

0/ 9
Low bleeding risk1.13%/year

Low bleeding risk. Anticoagulation is generally appropriate if CHA₂DS₂-VASc score warrants it. No specific risk-reduction interventions required.

Use alongside

CHA₂DS₂-VAScORBIT

Pisters R et al. A Novel User-Friendly Score (HAS-BLED) to Assess 1-Year Risk of Major Bleeding in Patients With Atrial Fibrillation. Chest. 2010;138(5):1093-1100.

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Cambridge Memorial Hospital logo
Jane Pauley Community Health Center logo
Sensible Care logo
Strive Health logo
New Brunswick Media Society logo
DMC Primary care logo
Airrosti logo
Wilmington Health logo
Cambridge Memorial Hospital logo
Jane Pauley Community Health Center logo
Sensible Care logo
Strive Health logo
New Brunswick Media Society logo
DMC Primary care logo
Airrosti logo
Wilmington Health logo
Cambridge Memorial Hospital logo
Jane Pauley Community Health Center logo
Sensible Care logo
Strive Health logo
New Brunswick Media Society logo
DMC Primary care logo
Airrosti logo
Heidi Tools

HAS-BLED Score Calculator

The HAS-BLED score is a validated clinical tool that estimates the 1-year risk of major bleeding in patients with atrial fibrillation (AF) being considered for oral anticoagulation. Calculate HAS-BLED below:

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HAS-BLED Score Calculator Key Points

Medical knowledge only. Not for autonomous decision making. Check sources and use your clinical judgement.

HAS-BLED scores guide anticoagulation in atrial fibrillation by flagging correctable bleeding risk factors. While some guidelines (ACCP, Canadian CCS) formally recommend its use, the 2024 ESC and 2023 ACC/AHA/ACCP/HRS guidelines no longer mandate any single bleeding score, noting poor discrimination and the risk of inappropriately withholding anticoagulation. Across all guidelines, one principle is universal: anticoagulation must not be stopped on the basis of bleeding risk factors alone.

A score of 3 or higher signals high bleeding risk and calls for more frequent clinical review after starting oral anticoagulation, not withdrawal of therapy. Clinicians act on the reversible components: control hypertension, stabilise a labile INR or switch to a DOAC, stop unnecessary NSAIDs or antiplatelet agents, and address alcohol excess (more than 8 drinks per week). A proton pump inhibitor should be considered in patients with high-risk upper gastrointestinal mucosal lesions on anticoagulation.

Because high bleeding risk often travels with high stroke risk, the score is read alongside CHA₂DS₂-VASc so that net clinical benefit drives the decision. Both risks should be reassessed regularly over time, not only at initiation. For non-valvular AF, DOACs are preferred over warfarin for their lower intracranial bleeding risk, though gastrointestinal bleeding risk varies by agent (apixaban and dabigatran carry a lower GI bleeding risk compared to rivaroxaban and edoxaban).

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